Snake venom tripeptide

Structure diagram of serpentin-like venom triplet peptide:

Snake venom tripeptide

Analytical standards,HPLC≥98%

Snake venom tripeptide

(2S) -beta-alanyl-L-prolyl-2,4-diamino-N-(phenylmethyl)butanamide acetate

Alias: (2S) -beta-alanyl-L-prolyl-2, 4-diamino-N -(benzyl) butyamido acetate; (2S) -beta-alanyl-L-prolyl-2,4-diamino-N-(phenylmethyl)butanamide acetate; SYN-AKE; snake-tripeptide; H – beta – Ala – Pro – Dab – NHBzl. 2 acoh; Snake tr

CAS number: 823202-99-9

Molecular formula: C19H29N5O3.2(C2H4O2)

Molecular weight: 495.58

Introduction:

Sequence: H-β-Ala-Pro-Dab-NH-Bzl

Appearance: white powder

About us:

A venom like peptide is a small peptide that mimics the activity of a snake venom toxin, and instead of snake venom, it is an active peptide that resembles the structure of a snake venom toxin. The venom peptide was five times more effective than botulinum in reducing wrinkles, and was found to reduce wrinkles by 52 percent after 28 days of use in humans

Benefits:

1. Anti-wrinkle and anti-aging

2. Improve skin quality

3. Face, neck and hand care products

4. Improve skin texture, make skin smooth and delicate, enhance complexion

5. It can enter the deep layer of skin, replenish collagen, and eliminate the traces left by years

6. Play the role of moisturizing and hydrating, make the skin tight and smooth, bright and beautiful

7. Tighten anti-wrinkle, re-tighten V-shaped face, and restore youthful skin

8. Prevent skin melanin production

Role:

The dipeptide diamino-butadibenzyl amide diacetate/venom like tripeptide is an effective smoothing and anti-crease active product by relaxing facial muscles in a manner consistent with Waglerin 1, the neuromuscular blocking compound of Temple Viper venom. Snake venom tripeptides act on postsynaptic membranes and are reversible antagonists of the muscle nicotinic acetylcholine receptor (nmAChR). The snake venom tripeptide binds to the ε subunit of nmAChR and blocks the binding of acetylcholine to the receptor, resulting in the blockade of the receptor. In the blocked state, sodium ions cannot be ingested and depolarized, and nerve excitation transmission is blocked, thus muscle relaxation.